Cause of Mesothelioma
Mesothelioma is a very rare and deadly form of cancer which is caused by exposure to asbestos. Asbestos fibers can be inhaled or swallowed and become embedded in the cells that line the lungs, heart, and abdominal organs, known as the mesothelium. This causes the development of cancerous cells which multiply at a much faster rate than healthy cells, forming tumors that put pressure on the affected organs and cause mesothelioma symptoms.
The Link between Asbestos and Mesothelioma
Scientific and medical research has proven overwhelmingly that asbestos is the primary cause of mesothelioma cancer. While the exact biological correlation between asbestos and mesothelioma is still under debate, it is known that asbestos causes cellular damage that can result in cancer. A number of theories exist as to the process that leads to cancer:
- Asbestos causes cells to produce oncoproteins, which are the molecules that cause otherwise healthy mesothelial cells to divide at an abnormally rapid rate, which leads to the onset of cancer.
- When asbestos fibers enter the system, they disrupt cellular structures responsible for normal cell division. The resulting cellular changes lead to cancer.
- Asbestos irritates and inflames the mesothelial cells that line organs, leading to the production of scar tissue and the cellular damage that leads to cancer.
- Asbestos absorbed into the system causes the production of molecules known as free radicals. These free radicals damage DNA, which leads to the mutation of cells to become cancerous.
Contact a Mesothelioma Lawyer
If you have been exposed to asbestos and now suffer from mesothelioma, asbestosis, lung cancer or another disease related to asbestos exposure, a mesothelioma lawyer from Shrader & Associates, LLP can help you pursue justice. You may be eligible to recover financial compensation for your illness, but you need to act quickly.
To learn more about mesothelioma and your possible eligibility for a mesothelioma lawsuit, contact Shrader & Associates, LLP today.